Fracture healing and callus innervation after peripheral nerve resection in rats
Postmortem studies have shown elevated expression of mitochondrial fission proteins in the prefrontal cortex of AD patients ( Given that mitochondrial bioenergetic alterations appear early in the disease course, they are considered potential primary events underlying synaptic failure, neuroinflammation, oxidative stress, and neuronal loss ( One hallmark of AD is the impaired removal of damaged mitochondria due to defective autophagy
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